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7‑epitaxol  (SAS institute)


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    Structured Review

    SAS institute 7‑epitaxol
    7‑Epitaxol, supplied by SAS institute, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/7%E2%80%91epitaxol/7+epitaxol/pmc09716209-86-4-38
    Average 90 stars, based on 1 article reviews
    7‑epitaxol - by Bioz Stars, 2026-09
    90/100 stars

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    Related Articles

    Concentration Assay:

    Article Title: 7‐Epitaxol induces apoptosis in cisplatin‐resistant head and neck squamous cell carcinoma via suppression of AKT and MAPK signalling
    Article Snippet: Regarding cell cycle regulation, 7‐Epitaxol was found to cause cell cycle arrest at the G2/M phase and reduce cell cycle rate at the G0/G1 phase in high concentration (50 and 100 nM in Cis‐SCC9 and 100 nM in Cis‐SAS).

    Incubation:

    Article Title: 7‐Epitaxol induces apoptosis in cisplatin‐resistant head and neck squamous cell carcinoma via suppression of AKT and MAPK signalling
    Article Snippet: Regarding cell cycle regulation, 7‐Epitaxol was found to cause cell cycle arrest at the G2/M phase and reduce cell cycle rate at the G0/G1 phase in high concentration (50 and 100 nM in Cis‐SCC9 and 100 nM in Cis‐SAS).

    Staining:

    Article Title: 7‐Epitaxol induces apoptosis in cisplatin‐resistant head and neck squamous cell carcinoma via suppression of AKT and MAPK signalling
    Article Snippet: Regarding cell cycle regulation, 7‐Epitaxol was found to cause cell cycle arrest at the G2/M phase and reduce cell cycle rate at the G0/G1 phase in high concentration (50 and 100 nM in Cis‐SCC9 and 100 nM in Cis‐SAS).

    Flow Cytometry:

    Article Title: 7‐Epitaxol induces apoptosis in cisplatin‐resistant head and neck squamous cell carcinoma via suppression of AKT and MAPK signalling
    Article Snippet: Regarding cell cycle regulation, 7‐Epitaxol was found to cause cell cycle arrest at the G2/M phase and reduce cell cycle rate at the G0/G1 phase in high concentration (50 and 100 nM in Cis‐SCC9 and 100 nM in Cis‐SAS).



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